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Cymbalta is an antidepressant. It is used to treat depression. It is also used to treat pain caused by complications of diabetes or pain caused by fibromyalgia.

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Cymbalta dosage compared to lexapro or tysabri. They reported that atropine, carbamazepine, or phenobarbital produced an increased likelihood of developing akathisia or drowsiness at low doses of either drug compared to lorazepam and midazolam. However, a retrospective study of patients whose drowsiness became severe after administration of one or more these drugs in adults with epilepsy (17% of whom had a history drug-induced drowsy states) revealed that this outcome was rare, occurring in only 15 of 17 cases, or 3 patients, after the first 5 doses. These patients did not achieve significant improvement in cognitive deficits. It appears that atropine, carbamazepine, and phenobarbital do not produce significant side effects when administered in the usual therapeutic range. Drugs that increase central nervous system (CNS) neurotransmitter activity—including psychostimulants and psychostimulant-induced central nervous system (CNS) dyskinesias (e.g., cocaine, methylphenidate; benzodiazepines)—can also impair cognitive performance and increase drowsiness. There is some overlap between CNS dyskinesias resulting from abuse and those produced by drug-induced drowsiness. However, their mechanisms of activation are different, and these factors may also have an effect on the development of this side effect. For example, drug-induced drowsiness is often related to inhibition of the GABA A receptor, which may be related to the development of clinical presentation drug and/or to the route of administration. Also, drugs that induce drowsiness in healthy individuals, e.g., those acting as a sedative or euphoriant, may also impair cognitive performance if they cause central nervous system stimulation of the motor cortex (i.e., by means other than the CNS). However, in individuals with underlying brain disorders, especially if they have difficulty in distinguishing between their own thoughts and those of others, these cognitive impairments are likely not caused by central CNS stimulation, and these drugs do not produce drowsiness. In a series of studies, Lebwohl et al. (18–22) found that drowsiness produced by benzodiazepines was largely due to impairment in memory, verbal, performance tasks, and attention, while it was due in part to impaired attention the environment with respect to central auditory processing as the result of drowsiness that occurred because Cymbalta 60 Pills 30mg $179 - $2.98 Per pill the drug-induced CNS arousal. These findings were broadly consistent with those reported by other researchers (e.g., 2, 10, 22). In many studies, these drowsiness effects were related to sedation or reduced alertness that were not fully reversible by lowering the dose of sedative or psychostimulating agent. In other studies, these effects were partially reversible with doses that elicited less sedation, or with doses of the sedative (e.g., pentobarbital) or hypnosis that caused less CNS arousal. In the most comprehensive comparison of these effects, Foeeler et al. (11) compared the effects of sedative drug propofol on performance, and reported that the drug markedly impaired cognitive performance and a small number of tasks in healthy volunteers but that it did not impair these measures in subjects with prior histories of drug-induced drowsiness. These studies and others indicate that the mechanism of drowsiness produced by sedative agents (e.g., sedation and amnestic effects) is different from that produced by CNS stimulants (e.g., amphetamine). Additionally, these drugs can elevate the levels of certain neurotransmitters (e.g., dopamine, norepinephrine, serotonin, and noradrenaline) that affect cognitive performance. When sedative agents can produce sedation, the adverse responses to drugs may be delayed. Sedation and amnesia may also be diminished, particularly with newer hypnotic drugs or of the class (3, 4). However, many of the drug-induced drowsiness-related adverse reactions can be predicted with certainty, making it difficult to identify a drug-related cause. For example, drowsy-producing drugs can produce a variety of adverse reactions after acute administration, such as dizziness, confusion, visual disturbances, and insomnia. In many cases, the adverse effects are seen only after the drug has been used for a reasonable time. Because these responses are not fully reversible by dose reduction, treatment with an antihistamine or other agents that attenuate the acute response may be necessary. Although adverse reactions to antihistamines as a group are generally characterized by an initial episode of drowsiness, subsequent effects may include tachypnea, headache, fatigue, and hypothermia (23, 24). Other adverse reactions can develop after prolonged use of an antihistamine. For example, in a retrospective analysis of 454 cases a central and.

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